The amyloidogenicity of the influenza virus PB1-derived peptide sheds light on its antiviral activity
Review articleOpen access
Abstract:

Highlights•PB1(6–13) peptide is prone to form the amyloid-like fibrils.•PB1(6–13) is able to interact with influenza A virus PB1 N-terminus in peptide model.•Peptide induces the conformational transition in PB1 N-terminus in peptide model.•The results allow us to hypothesize a mechanism of PB1(6–13) antiviral action.

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